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mitosox red probe oxidation  (Thermo Fisher)


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    Structured Review

    Thermo Fisher mitosox red probe oxidation
    Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of <t>mitochondrial</t> <t>ROS</t> production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using <t>MitoSOX</t> red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.
    Mitosox Red Probe Oxidation, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mitosox+red+probe+oxidation/mitosox+red+probe+oxidation/pmc09314546-338-9-13
    Average 90 stars, based on 1 article reviews
    mitosox red probe oxidation - by Bioz Stars, 2026-09
    90/100 stars

    Images

    1) Product Images from "Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy"

    Article Title: Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy

    Journal: Cell Reports

    doi: 10.1016/j.celrep.2022.111124

    Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of mitochondrial ROS production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using MitoSOX red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.
    Figure Legend Snippet: Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of mitochondrial ROS production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using MitoSOX red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.

    Techniques Used: Derivative Assay


    Figure Legend Snippet:

    Techniques Used: Recombinant, Enzyme-linked Immunosorbent Assay, Plasmid Preparation, Software



    Similar Products

    90
    Thermo Fisher mitosox red probe oxidation
    Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of <t>mitochondrial</t> <t>ROS</t> production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using <t>MitoSOX</t> red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.
    Mitosox Red Probe Oxidation, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mitosox+red+probe+oxidation/mitosox+red+probe+oxidation/pmc09314546-338-9-13
    Average 90 stars, based on 1 article reviews
    mitosox red probe oxidation - by Bioz Stars, 2026-09
    90/100 stars
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    Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of mitochondrial ROS production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using MitoSOX red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.

    Journal: Cell Reports

    Article Title: Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy

    doi: 10.1016/j.celrep.2022.111124

    Figure Lengend Snippet: Complex I deficiency leads to altered mitochondrial function in LHON-affected individuals, which is compensated in carriers (A–D) Measurements of mitochondrial ROS production in LHON-derived 3460 fibroblasts (A) and cybrids (B) and in 11778 fibroblasts (C) and cybrids (D) by using MitoSOX red probe. (E–H) The mitochondrial transmembrane potential (Ψ m ) of 3460 and 11778 fibroblasts and cybrids was detected by using the Ψ m -sensitive probe TMRM. When indicated, the cells were STARV. for 30 min before TMRM loading. Data are presented as means ± SEM. n = at least 5 visual fields per at least 3 independent samples per condition. ∗ p < 0.05, ∗∗ p < 0.01, and ∗∗∗ p < 0.001.

    Article Snippet: Total ROS release from mitochondria was estimated fluorometrically by MitoSOX Red probe oxidation (Thermo Fisher and TaliTM Image-Based Cytometer).

    Techniques: Derivative Assay

    Journal: Cell Reports

    Article Title: Pathological mitophagy disrupts mitochondrial homeostasis in Leber’s hereditary optic neuropathy

    doi: 10.1016/j.celrep.2022.111124

    Figure Lengend Snippet:

    Article Snippet: Total ROS release from mitochondria was estimated fluorometrically by MitoSOX Red probe oxidation (Thermo Fisher and TaliTM Image-Based Cytometer).

    Techniques: Recombinant, Enzyme-linked Immunosorbent Assay, Plasmid Preparation, Software